Turmeric is one the most thoroughly researched plants in existence today. Its medicinal properties and components (primarily curcumin) have been the subject of over 5600 peer-reviewed and published biomedical studies. In fact, our five-year long research project on this sacred plant has revealed over 600 potential preventive and therapeutic applications, as well as 175 distinct beneficial physiological effects. This entire database of 1,585 ncbi-hyperlinked turmeric abstracts can be downloaded as a PDF at our Downloadable Turmeric Document page, and acquired either as a retail item or with 200 GMI-tokens, for those of you who are already are members and receive them automatically each month.
Given the sheer density of research performed on this remarkable spice, it is no wonder that a growing number of studies have concluded that it compares favorably to a variety of conventional medications, including:
- Lipitor/Atorvastatin(cholesterol medication): [For additional information see: curcumin and ‘high cholesterol’ research – 8 abstracts]
- Corticosteroids (steroid medications): [for additional curcumin and inflammation research – 52 abstracts]
- Prozac/Fluoxetine & Imipramine (antidepressants): [for additional curcumin and depression research – 5 abstracts]
- Aspirin (blood thinner): [for additional curcumin and anti-platelet research]
- Anti-inflammatory Drugs: [for additional curcumin and anti-proliferative research – 15 abstracts]
- Oxaliplatin (chemotherapy drug): [for additional curcumin and colorectal cancer research – 52 abstracts]
- Metformin (diabetes drug): A 2009 study published in the journal Biochemitry and Biophysical Research Community explored how curcumin might be valuable in treating diabetes, finding that it activates AMPK (which increases glucose uptake) and suppresses gluconeogenic gene expression (which suppresses glucose production in the liver) in hepatoma cells. Interestingly, they found curcumin to be 500 times to 100,000 times (in the form known as tetrahydrocurcuminoids(THC)) more potent than metformin in activating AMPK and its downstream target acetyl-CoA carboxylase (ACC). [ix]
See the article on GreeMedInfo.com for all the details
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